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Copper Kills CORVID-19

Updated: Mar 27, 2020


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For more than 5,o00 years ancient civilizations have referred to copper as a symbol of eternal life, a material that cultures across the globe have recognized as vital to our health.


Colloidal Copper can effectively help to prevent the spread of respiratory viruses, which are linked to previous outbreaks of severe acute respiratory syndrome (SARS) and Middle East respiratory syndrome (MERS), research has shown. Animal coronaviruses that 'host jump' to humans, such as SARS and MERS, result in severe infections with high mortality. Researchers found that a closely-related human coronavirus - CORVID-19 - can remain infectious on common surface materials for hours to several days, but is rapidly destroyed on copper.


Human coronavirus, which also has ancestral links with bat-like viruses responsible for SARS and MERS, was found to be permanently and rapidly deactivated upon contact with copper. What's more, the viral genome and structure of the viral particles were destroyed, so nothing remained that could pass on an infection. With the lack of antiviral treatments, copper offers a measure that can help reduce the risk of these infections spreading.


Professor Bill Keevil, co-author and Chair in Environmental Healthcare at the University of Southampton, said: "Respiratory viruses are responsible for more deaths, globally, than any other infectious agent. The evolution of new respiratory viruses, and the re-emergence of historic virulent strains, poses a significant threat to human health.

"The rapid inactivation and irreversible destruction of the virus observed on copper and copper alloy surfaces suggests that the incorporation of copper alloy surfaces -- in conjunction with effective cleaning regimes and good clinical practice -- could help control transmission of these viruses."


Previous research by Professor Keevil and Dr Warnes has proved copper's efficacy against norovirus, influenza and hospital superbugs, such as MRSA and Klebsiella, plus stopping the transfer of antibiotic resistance genes to other bacteria to create new superbugs.



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